
Peptide · Vial
Verified recordBPC 157
$90An experimental peptide best known for early research into tissue repair, tendon injury and wound response.

EternaGen / Launch batch pendingPeptide · Research use only
A tuftsin-derived heptapeptide explored in stress-response, GABA-related and intranasal neurobiology research.
Purchasing review required
Can’t find the right record?Send support the product name or batch code.
Contact supportProduct information
A tuftsin-derived heptapeptide explored in stress-response, GABA-related and intranasal neurobiology research.
Selank has a small comparative human study and a wider mechanistic literature, much of it from animal or laboratory models. Independent replication, route-specific safety and clinically meaningful outcomes remain insufficiently established.
Selank is a synthetic seven-amino-acid peptide with the sequence Thr-Lys-Pro-Arg-Pro-Gly-Pro. It is derived from the immunomodulatory peptide tuftsin and appears in a limited body of human, animal and laboratory research.Specifications
The batch code, analytical results and handling specifications will publish after verification.
This catalogue entry describes a research material, not an approved therapeutic formulation. Published findings do not provide directions for personal use and cannot verify the safety or performance of a separately sourced material.
Explore the research
Check what was studied, what researchers observed, and the limitation attached to each finding.
A 62-person comparative study reported psychometric outcomes for Selank and medazepam in generalized anxiety disorder and neurasthenia.
Evidence so far: Small single-study human evidenceLaboratory and animal work has examined GABA- and NMDA-receptor binding as possible explanatory pathways.
Evidence so far: Mechanistic and animal evidenceThe human evidence base is narrow, regionally concentrated and insufficient to establish broad efficacy or long-term safety.
Evidence so far: Major evidence gapProposed mechanism
Laboratory studies have examined Selank in relation to enkephalin degradation and GABA-receptor binding. Animal experiments suggest that administration route and strain can materially change the observed signal, which limits broad interpretation.
Study records
See what researchers tested, what they observed and the limitation attached to each result.
Evidence gaps
Primary sources
Research catalogue
Before entering EternaGen, confirm both statements.
By entering, you affirm that both statements above are true.
Unable to confirm? Leave site